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Eken, Ahmet; Cansever, Murat; Somekh, Ido; Mizoguchi, Yoko; Zietara, Natalia; Okus, Fatma Zehra; Erdem, Serife; Canatan, Halit; Akyol, Sefika; Ozcan, Alper; Karakukcu, Musa; Hollizeck, Sebastian; Rohlfs, Meino; Unal, Ekrem; Klein, Christoph; Patiroglu, Turkan
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Munich, Germany</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Okus, Fatma Zehra</subfield> <subfield code="u">Erciyes Univ, Genome & Stem Cell Ctr GENKOK, Dept Med Biol, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Erdem, Serife</subfield> <subfield code="u">Erciyes Univ, Genome & Stem Cell Ctr GENKOK, Dept Med Biol, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Canatan, Halit</subfield> <subfield code="u">Erciyes Univ, Genome & Stem Cell Ctr GENKOK, Dept Med Biol, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Akyol, Sefika</subfield> <subfield code="u">Erciyes Univ, Div Pediat Hematol Oncol, Dept Pediat, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Ozcan, Alper</subfield> <subfield code="u">Erciyes Univ, Div Pediat Hematol Oncol, Dept Pediat, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Karakukcu, Musa</subfield> <subfield code="u">Erciyes Univ, Div Pediat Hematol Oncol, Dept Pediat, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Hollizeck, Sebastian</subfield> <subfield code="u">Ludwig Maximilians Univ Munchen, Univ Hosp, Dept Pediat, Dr von Hauner Childrens Hosp, Munich, Germany</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Rohlfs, Meino</subfield> <subfield code="u">Ludwig Maximilians Univ Munchen, Univ Hosp, Dept Pediat, Dr von Hauner Childrens Hosp, Munich, Germany</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Unal, Ekrem</subfield> <subfield code="u">Erciyes Univ, Div Pediat Hematol Oncol, Dept Pediat, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Klein, Christoph</subfield> <subfield code="u">Ludwig Maximilians Univ Munchen, Univ Hosp, Dept Pediat, Dr von Hauner Childrens Hosp, Munich, Germany</subfield> </datafield> <datafield tag="700" ind1=" " ind2=" "> <subfield code="a">Patiroglu, Turkan</subfield> <subfield code="u">Erciyes Univ, Div Pediat Hematol Oncol, Dept Pediat, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="909" ind1="C" ind2="4"> <subfield code="p">JOURNAL OF CLINICAL IMMUNOLOGY</subfield> <subfield code="v">39</subfield> <subfield code="n">4</subfield> <subfield code="c">391-400</subfield> </datafield> <datafield tag="980" ind1=" " ind2=" "> <subfield code="a">user-tubitak-destekli-proje-yayinlari</subfield> </datafield> <datafield tag="540" ind1=" " ind2=" "> <subfield code="a">Creative Commons Attribution</subfield> <subfield code="u">http://www.opendefinition.org/licenses/cc-by</subfield> </datafield> <datafield tag="024" ind1=" " ind2=" "> <subfield code="a">10.1007/s10875-019-00632-5</subfield> <subfield code="2">doi</subfield> </datafield> <datafield tag="245" ind1=" " ind2=" "> <subfield code="a">Genetic Deficiency and Biochemical Inhibition of ITK Affect Human Th17, Treg, and Innate Lymphoid Cells</subfield> </datafield> <datafield tag="100" ind1=" " ind2=" "> <subfield code="a">Eken, Ahmet</subfield> <subfield code="u">Erciyes Univ, Genome & Stem Cell Ctr GENKOK, Dept Med Biol, Fac Med, TR-38030 Kayseri, Turkey</subfield> </datafield> <datafield tag="909" ind1="C" ind2="O"> <subfield code="o">oai:zenodo.org:74957</subfield> <subfield code="p">user-tubitak-destekli-proje-yayinlari</subfield> </datafield> <datafield tag="650" ind1="1" ind2="7"> <subfield code="2">opendefinition.org</subfield> <subfield code="a">cc-by</subfield> </datafield> <datafield tag="260" ind1=" " ind2=" "> <subfield code="c">2019-01-01</subfield> </datafield> <datafield tag="856" ind1="4" ind2=" "> <subfield code="u">https://aperta.ulakbim.gov.trrecord/74957/files/bib-77f52cd3-043f-4c35-abd7-d7f2aa92a5ef.txt</subfield> <subfield code="z">md5:f7b034a7cbbeaf8e0df20858d09bab2a</subfield> <subfield code="s">358</subfield> </datafield> <datafield tag="542" ind1=" " ind2=" "> <subfield code="l">open</subfield> </datafield> <controlfield tag="005">20210316041835.0</controlfield> <controlfield tag="001">74957</controlfield> <datafield tag="980" ind1=" " ind2=" "> <subfield code="a">publication</subfield> <subfield code="b">article</subfield> </datafield> <datafield tag="520" ind1=" " ind2=" "> <subfield code="a">Purpose Interleukin-2-inducible T cell kinase (ITK) is an important mediator of T cell receptor signaling. Loss of function mutations in ITK results in hypogammaglobulinemia and CD4+ T cell loss in humans, and the patients often present with EBV-associated B cell lymphoproliferative syndrome. Itk-deficient mice show loss of T cell naivety, impaired cytolytic activity of CD8+ T cells, and defects in CD4+ T cell lineage choice decisions. In mice, Itk mutations were shown to affect Th17-Treg lineage choice in favor of the latter. In this study, we explored whether human ITK reciprocally regulates Th17-Treg balance as its murine ortholog.</subfield> </datafield> </record>
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