1 Ocak 2017 Dergi makalesi Açık Erişim

Turan, Ihsan; Hutchins, B. Ian; Hacihamdioglu, Bulent; Kotan, L. Damla; Gurbuz, Fatih; Ulubay, Ayca; Mengen, Eda; Yuksel, Bilgin; Wray, Susan; Topaloglu, A. Kemal

Dublin Core

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  <dc:creator>Turan, Ihsan</dc:creator>
  <dc:creator>Hutchins, B. Ian</dc:creator>
  <dc:creator>Hacihamdioglu, Bulent</dc:creator>
  <dc:creator>Kotan, L. Damla</dc:creator>
  <dc:creator>Gurbuz, Fatih</dc:creator>
  <dc:creator>Ulubay, Ayca</dc:creator>
  <dc:creator>Mengen, Eda</dc:creator>
  <dc:creator>Yuksel, Bilgin</dc:creator>
  <dc:creator>Wray, Susan</dc:creator>
  <dc:creator>Topaloglu, A. Kemal</dc:creator>
  <dc:date>2017-01-01</dc:date>
  <dc:description>Context: Gonadotropin-releasing hormone neurons originate outside the central nervous systemin the olfactory placode and migrate into the central nervous system, becoming integral components of the hypothalamic-pituitary-gonadal axis. Failure of this migration can lead to idiopathic hypogonadotropic hypogonadism (IHH)/Kallmann syndrome (KS). We have previously shown that CCDC141 knockdown leads to impaired migration of GnRH neurons but not of olfactory receptor neurons.</dc:description>
  <dc:identifier>https://aperta.ulakbim.gov.trrecord/46747</dc:identifier>
  <dc:identifier>oai:zenodo.org:46747</dc:identifier>
  <dc:rights>info:eu-repo/semantics/openAccess</dc:rights>
  <dc:rights>http://www.opendefinition.org/licenses/cc-by</dc:rights>
  <dc:source>JOURNAL OF CLINICAL ENDOCRINOLOGY &amp; METABOLISM 102(6) 1816-1825</dc:source>
  <dc:title>CCDC141 Mutations in Idiopathic Hypogonadotropic Hypogonadism</dc:title>
  <dc:type>info:eu-repo/semantics/article</dc:type>
  <dc:type>publication-article</dc:type>
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