1 Ocak 2018 Dergi makalesi Açık Erişim

Balcik-Ercin, Pelin; Cetin, Metin; Yalim-Camci, Irem; Odabas, Gorkem; Tokay, Nurettin; Sayan, A. Emre; Yagci, Tamer

Dublin Core

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  <dc:creator>Balcik-Ercin, Pelin</dc:creator>
  <dc:creator>Cetin, Metin</dc:creator>
  <dc:creator>Yalim-Camci, Irem</dc:creator>
  <dc:creator>Odabas, Gorkem</dc:creator>
  <dc:creator>Tokay, Nurettin</dc:creator>
  <dc:creator>Sayan, A. Emre</dc:creator>
  <dc:creator>Yagci, Tamer</dc:creator>
  <dc:date>2018-01-01</dc:date>
  <dc:description>ZEB2 is a transcriptional repressor that regulates epithelial-to-mesenchymal transition (EMT) through binding to bipartite E-box motifs in gene regulatory regions. Despite the abundant presence of E-boxes within the human genome and the multiplicity of pathophysiological processes regulated during ZEB2-induced EMT, only a small fraction of ZEB2 targets has been identified so far. Hence, we explored genome-wide ZEB2 binding by chromatin immunoprecipitation-sequencing (ChIP-seq) under endogenous ZEB2 expression conditions.</dc:description>
  <dc:identifier>https://aperta.ulakbim.gov.trrecord/36631</dc:identifier>
  <dc:identifier>oai:zenodo.org:36631</dc:identifier>
  <dc:rights>info:eu-repo/semantics/openAccess</dc:rights>
  <dc:rights>http://www.opendefinition.org/licenses/cc-by</dc:rights>
  <dc:source>CELLULAR ONCOLOGY 41(4) 379-393</dc:source>
  <dc:title>Genome-wide analysis of endogenously expressed ZEB2 binding sites reveals inverse correlations between ZEB2 and GalNAc-transferase GALNT3 in human tumors</dc:title>
  <dc:type>info:eu-repo/semantics/article</dc:type>
  <dc:type>publication-article</dc:type>
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