1 Ocak 2019 Dergi makalesi Açık Erişim

Kizilgul, M.; Wilhelm, J. J.; Dunn, T. B.; Beilman, G. J.; Pruett, T. L.; Chinnakotla, S.; Amin, K.; Hering, B. J.; Bellin, M. D.

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  <identifier identifierType="URL">https://aperta.ulakbim.gov.tr/record/75609</identifier>
  <creators>
    <creator>
      <creatorName>Kizilgul, M.</creatorName>
      <givenName>M.</givenName>
      <familyName>Kizilgul</familyName>
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    <creator>
      <creatorName>Wilhelm, J. J.</creatorName>
      <givenName>J. J.</givenName>
      <familyName>Wilhelm</familyName>
      <affiliation>Univ Minnesota, Schulze Diabet Inst, Minneapolis, MN USA</affiliation>
    </creator>
    <creator>
      <creatorName>Dunn, T. B.</creatorName>
      <givenName>T. B.</givenName>
      <familyName>Dunn</familyName>
      <affiliation>Univ Minnesota, Dept Surg, Box 242 UMHC, Minneapolis, MN 55455 USA</affiliation>
    </creator>
    <creator>
      <creatorName>Beilman, G. J.</creatorName>
      <givenName>G. J.</givenName>
      <familyName>Beilman</familyName>
      <affiliation>Univ Minnesota, Dept Surg, Box 242 UMHC, Minneapolis, MN 55455 USA</affiliation>
    </creator>
    <creator>
      <creatorName>Pruett, T. L.</creatorName>
      <givenName>T. L.</givenName>
      <familyName>Pruett</familyName>
      <affiliation>Univ Minnesota, Dept Surg, Box 242 UMHC, Minneapolis, MN 55455 USA</affiliation>
    </creator>
    <creator>
      <creatorName>Chinnakotla, S.</creatorName>
      <givenName>S.</givenName>
      <familyName>Chinnakotla</familyName>
      <affiliation>Univ Minnesota, Dept Surg, Box 242 UMHC, Minneapolis, MN 55455 USA</affiliation>
    </creator>
    <creator>
      <creatorName>Amin, K.</creatorName>
      <givenName>K.</givenName>
      <familyName>Amin</familyName>
      <affiliation>Univ Minnesota, Dept Lab Med &amp; Pathol, Minneapolis, MN 55455 USA</affiliation>
    </creator>
    <creator>
      <creatorName>Hering, B. J.</creatorName>
      <givenName>B. J.</givenName>
      <familyName>Hering</familyName>
      <affiliation>Univ Minnesota, Schulze Diabet Inst, Minneapolis, MN USA</affiliation>
    </creator>
    <creator>
      <creatorName>Bellin, M. D.</creatorName>
      <givenName>M. D.</givenName>
      <familyName>Bellin</familyName>
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  <titles>
    <title>The Prognostic Significance Of Glutamic Acid Decarboxylase Antibodies In Patients With Chronic Pancreatitis Undergoing Total Pancreatectomy With Islet Autotransplantation</title>
  </titles>
  <publisher>Aperta</publisher>
  <publicationYear>2019</publicationYear>
  <dates>
    <date dateType="Issued">2019-01-01</date>
  </dates>
  <resourceType resourceTypeGeneral="Text">Journal article</resourceType>
  <alternateIdentifiers>
    <alternateIdentifier alternateIdentifierType="url">https://aperta.ulakbim.gov.tr/record/75609</alternateIdentifier>
  </alternateIdentifiers>
  <relatedIdentifiers>
    <relatedIdentifier relatedIdentifierType="DOI" relationType="IsIdenticalTo">10.1016/j.diabet.2018.01.001</relatedIdentifier>
  </relatedIdentifiers>
  <rightsList>
    <rights rightsURI="http://www.opendefinition.org/licenses/cc-by">Creative Commons Attribution</rights>
    <rights rightsURI="info:eu-repo/semantics/openAccess">Open Access</rights>
  </rightsList>
  <descriptions>
    <description descriptionType="Abstract">Aim. - Islet autotransplantation (IAT) is considered a 'non-immune' model of islet transplant, with no risk for autoimmune-mediated beta cell loss, but we have previously observed de novo type 1 diabetes in one total pancreatectomy with islet autotransplantation (TPIAT) recipient. We aimed to investigate the clinical significance of glutamic acid decarboxylase antibodies (GADA), as a sensitive marker for autoimmune diabetes mellitus (DM), in patients with chronic pancreatitis undergoing TPIAT.</description>
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