1 Ocak 2018 Dergi makalesi Açık Erişim

Cakmak, Hakan; Seval-Celik, Yasemin; Arlier, Sefa; Guzeloglu-Kayisli, Ozlem; Schatz, Frederick; Arici, Aydin; Kayisli, Umit A.

DataCite XML

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  <identifier identifierType="URL">https://aperta.ulakbim.gov.tr/record/30943</identifier>
  <creators>
    <creator>
      <creatorName>Cakmak, Hakan</creatorName>
      <givenName>Hakan</givenName>
      <familyName>Cakmak</familyName>
    </creator>
    <creator>
      <creatorName>Seval-Celik, Yasemin</creatorName>
      <givenName>Yasemin</givenName>
      <familyName>Seval-Celik</familyName>
    </creator>
    <creator>
      <creatorName>Arlier, Sefa</creatorName>
      <givenName>Sefa</givenName>
      <familyName>Arlier</familyName>
      <affiliation>Univ S Florida, Dept Obstet &amp; Gynecol, Morsani Coll Med, 4202 E Fowler Ave, Tampa, FL 33620 USA</affiliation>
    </creator>
    <creator>
      <creatorName>Guzeloglu-Kayisli, Ozlem</creatorName>
      <givenName>Ozlem</givenName>
      <familyName>Guzeloglu-Kayisli</familyName>
      <affiliation>Univ S Florida, Dept Obstet &amp; Gynecol, Morsani Coll Med, 4202 E Fowler Ave, Tampa, FL 33620 USA</affiliation>
    </creator>
    <creator>
      <creatorName>Schatz, Frederick</creatorName>
      <givenName>Frederick</givenName>
      <familyName>Schatz</familyName>
      <affiliation>Univ S Florida, Dept Obstet &amp; Gynecol, Morsani Coll Med, 4202 E Fowler Ave, Tampa, FL 33620 USA</affiliation>
    </creator>
    <creator>
      <creatorName>Arici, Aydin</creatorName>
      <givenName>Aydin</givenName>
      <familyName>Arici</familyName>
      <affiliation>Yale Univ, Sch Med, Dept Obstet Gynecol &amp; Reprod Sci, New Haven, CT USA</affiliation>
    </creator>
    <creator>
      <creatorName>Kayisli, Umit A.</creatorName>
      <givenName>Umit A.</givenName>
      <familyName>Kayisli</familyName>
      <affiliation>Univ S Florida, Dept Obstet &amp; Gynecol, Morsani Coll Med, 4202 E Fowler Ave, Tampa, FL 33620 USA</affiliation>
    </creator>
  </creators>
  <titles>
    <title>P38 Mitogen-Activated Protein Kinase Is Involved In The Pathogenesis Of Endometriosis By Modulating Inflammation, But Not Cell Survival</title>
  </titles>
  <publisher>Aperta</publisher>
  <publicationYear>2018</publicationYear>
  <dates>
    <date dateType="Issued">2018-01-01</date>
  </dates>
  <resourceType resourceTypeGeneral="Text">Journal article</resourceType>
  <alternateIdentifiers>
    <alternateIdentifier alternateIdentifierType="url">https://aperta.ulakbim.gov.tr/record/30943</alternateIdentifier>
  </alternateIdentifiers>
  <relatedIdentifiers>
    <relatedIdentifier relatedIdentifierType="DOI" relationType="IsIdenticalTo">10.1177/1933719117725828</relatedIdentifier>
  </relatedIdentifiers>
  <rightsList>
    <rights rightsURI="http://www.opendefinition.org/licenses/cc-by">Creative Commons Attribution</rights>
    <rights rightsURI="info:eu-repo/semantics/openAccess">Open Access</rights>
  </rightsList>
  <descriptions>
    <description descriptionType="Abstract">Background: Local pro-inflammatory environment and enhanced cell survival contribute to the endometriosis development. A serine/threonine kinase p38 mitogen-activated protein kinase (MAPK) mediates intracellular signaling of cytokine production, cell proliferation, and apoptosis in different cell types. The current study compares p38 MAPK activity in normal endometrium and endometriosis, and assesses role(s) of p38 MAPK on cytokine production and cell survival in endometriosis.</description>
  </descriptions>
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