1 Ocak 2021 Dergi makalesi Açık Erişim

Yildirim, Caner; Ozkaya, Beytullah; Bal, Ramazan

Dublin Core

<?xml version='1.0' encoding='utf-8'?>
<oai_dc:dc xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
  <dc:creator>Yildirim, Caner</dc:creator>
  <dc:creator>Ozkaya, Beytullah</dc:creator>
  <dc:creator>Bal, Ramazan</dc:creator>
  <dc:date>2021-01-01</dc:date>
  <dc:description>ATP-sensitive potassium (K-ATP) channels and transient receptor potential melastatin 2 (TRPM2) channels are commonly expressed both pre- and postsynaptically in the central nervous system (CNS). We hypothesized that K-ATP and TRPM2 may couple metabolic status to the resting membrane potential of octopus neurons of the mouse ventral cochlear nucleus (VCN).</dc:description>
  <dc:identifier>https://aperta.ulakbim.gov.trrecord/238026</dc:identifier>
  <dc:identifier>oai:aperta.ulakbim.gov.tr:238026</dc:identifier>
  <dc:rights>info:eu-repo/semantics/openAccess</dc:rights>
  <dc:rights>http://www.opendefinition.org/licenses/cc-by</dc:rights>
  <dc:source>BRAIN RESEARCH BULLETIN 170 115-128</dc:source>
  <dc:title>KATP and TRPM2-like channels couple metabolic status to resting membrane potential of octopus neurons in the mouse ventral cochlear nucleus</dc:title>
  <dc:type>info:eu-repo/semantics/article</dc:type>
  <dc:type>publication-article</dc:type>
</oai_dc:dc>