Published January 1, 2018 | Version v1
Journal article Open

Potential effects of metformin in DNA BER system based on oxidative status in type 2 diabetes

  • 1. Ufuk Univ, Med Biol Dept, TR-06520 Ankara, Turkey
  • 2. Ufuk Univ, Med Biochem Dept, TR-06520 Ankara, Turkey
  • 3. Ufuk Univ, Med Genet Dept, TR-06520 Ankara, Turkey
  • 4. Ufuk Univ, Internal Med Dept, TR-06520 Ankara, Turkey

Description

Metformin is used to reduce hyperglycemia that induces energetic stress and leads to reduction in gluconeogenesis. Also, metformin inhibits complex I in oxidative phosphorylation, thereby decreasing cellular ATP levels. Activation of AMPK by the reduced ATP levels can induce inhibition of reactive oxygen species (ROS) production and activate p53-mediated DNA repair.

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