Yayınlanmış 1 Ocak 2018
| Sürüm v1
Dergi makalesi
Açık
A SGLT2 inhibitor dapagliflozin suppresses prolonged ventricular-repolarization through augmentation of mitochondrial function in insulin-resistant metabolic syndrome rats
Oluşturanlar
- 1. Ankara Univ, Dept Biophys, Fac Med, Ankara, Turkey
- 2. Ankara Univ, Fac Med, Internal Med, Ankara, Turkey
Açıklama
Background: Metabolic syndrome (MetS) is a prevalent risk factor for cardiac dysfunction. Although SGLT2-inhibitors have important cardioprotective effects in hyperglycemia, their underlying mechanisms are complex and not completely understood. Therefore, we examined mechanisms of a SGLT2-inhibitor dapagliflozin (DAPA)-related cardioprotection in overweight insulin-resistant MetS-rats comparison with insulin (INSU), behind its glucose-lowering effect.
Dosyalar
bib-0ac2322c-81e6-44a3-b1dc-360da49dbbe9.txt
Dosyalar
(290 Bytes)
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